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Caseous necrosis
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This type of necrosis occurs as part of granulomatous inflammation and is a manifestation of partial immunity caused by the interaction of T lymphocytes (CD4+, CD8+, and CD4− CD8−), macrophages, and probably cytokines, such as interferon-γ, derived from these cells.
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Tuberculosis is the leading cause of caseous necrosis.
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Caseous necrosis combines features of both coagulative necrosis and liquefactive necrosis.
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On gross examination, caseous necrosis has a cheese-like (caseous) consistency.
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On histologic examination, caseous necrosis has an amorphous eosinophilic appearance.
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Gangrenous necrosis
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This type of necrosis most often affects the lower extremities or bowel and is secondary to vascular occlusion.
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When complicated by infective heterolysis and consequent liquefactive necrosis, gangrenous necrosis is called wet gangrene.
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When characterized primarily by coagulative necrosis without liquefaction, gangrenous necrosis is called dry gangrene.
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Fibrinoid necrosis
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Fat necrosis occurs in two forms:
https://www.andreasastier.com/blog/the-different-types-of-necrosis-and-their-histological-identifications
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